| Sound Process |
Brief Description |
A Glimpse – "who does what" in Sound Production |
Examples of "Breakdowns" that Affect Sound Process |
Possible Symptoms |
Generating Air Pressure |
"Breath Support"
Coordinated
functions of diaphragm, abdominal and chest muscles, lungs and chest
cavity move air column upwards through vocal folds |
Diaphragm, abdominal and chest muscles move air into and out of lungs
Lungs are the organs for air
Bronchi and trachea are the passageway for air from lungs |
Lung disease
Airway obstruction: asthma, subglottic stenosis
Paresis/paralysis of muscles |
Shortness of breath
Weak voice |
Glottic Closure
Position of vocal folds |
Vocal Fold Position Critical
Vocal folds are in closed position for speech or singing
[Note: Vocal folds are open for breathing] |
Laryngeal muscles contract to close vocal folds
Arytenoid cartilages pivot to move vocal folds towards midline ["closed"]
RLN and SLN bring nerve inputs to muscles
Vocal fold mass and edge contribute to glottic closure |
Paresis/paralysis – RLN/SLN
Muscle weakness
Neurological diseases; muscular disorders
Arytenoid arthritis
Vocal fold granuloma
Vocal fold nodules, polyps, cysts
Vocal fold atrophy
Vocal fold scarring |
Hoarseness
Breathiness
Effortful phonation
Vocal fatigue
Diplophonia |
Vocal Fold Vibration Produces Voiced Sound
|
Wavelike Vibration from Bottom to Top–repeat vibratory cycles
With each vocal fold vibratory cycle, a puff of air escapes, producing voice sound ("buzzy sound")
Singing voice produces unique sound spectra (singing formants) that are distinct from spoken voice |
Superficial lamina propria is the main vibrating layer in vocal fold mucosa
Vocal fold pliability is critical for vibration
Vocal fold surface integrity allows mucosal wave propagation
Vocal fold mass and edge contribute to glottic closure |
Vocal fold scar
Vocal fold lesions: cysts, nodules, polyps, papilloma
Vocal fold granuloma
Swelling and inflammation (reflux laryngitis, viral laryngitis)
Reinke's edema
Paresis/paralysis
Hemorrhage, vascular ectasias |
Hoarseness
Effortful phonation
Weak voice
Speaking voice lower than usual
"Vocal fry"
Voice fatigue |
|
Amplitude of Sound Waves
Loudness is achieved by:
Increasing air pressure/flow
Increasing vocal fold resistance |
Breath support
Laryngeal muscles contract to adjust tension of vocal folds
Vocal fold elasticity allows folds to "open wider" and "stay apart" longer |
Vocal fold scar
Paresis/paralysis
Vocal fold lesions: cysts, nodules, polyps, papilloma
Vocal fold granuloma
Swelling and inflammation (reflux laryngitis, viral laryngitis) |
Unable to project voice
Weak voice
Voice breaks |
Voice Pitch or Highness / Lowness |
Frequency of Sound Waves
Increased tension for high notes (high frequency)
Decreased tension for low notes (low frequency)
Greater air pressure and increased tension for high notes |
Laryngeal muscles contract to adjust tension of vocal folds–especially for high notes
Vocal fold elasticity/pliability
Vocal fold edge integrity |
SLN paresis/paralysis
Vocal fold scar
Reinke's edema
Vocal fold lesions |
Unable to hit high notes
Loss of glissando
Voice breaks |
Voice Quality or Timbre |
Person's "Own" Voice
Vocal tract resonators amplify and modify voiced sound, giving personal quality to a person's voice
e.g., John's voice not equal to Jim's voice |
Vocal fold elasticity/pliability
Vocal fold resistance
Vocal fold mass
Person's vocal fold features: e.g., stiffness, bulk, size
Vocal tract resonators (throat, oral cavity, nasal cavities) |
Vocal fold scar
Vocal fold inflammation
Reinke's Edema
Common cold |
Hoarseness, etc.
Off expected pitch
Nasal quality to voice |